Individuals who are tired from a lack of sleep will typically want to eat extra food the following day so as to get energy from the food. As time goes along the extra calories are likely to be stored as fat, resulting in weight gain and eventual obesity.
The new science paints a clear picture of changes in brain chemistry that actual start before weight is gained. It is a combination of the arousal thermostat set too high and leptin resistance (high leptin in the blood that is not getting into the brain properly)
Hypocretin neurons that comprising the arousal thermostat contain leptin receptors, meaning that leptin speaks directly to them so as to slow them down. When leptin is not entering the brain properly then there is a lack of instruction to not eat. This causes the over-stimulated hyporcretin system to desire excess food, a misguided signal left over from the days of the hunter-gather when extra mental vigilance and less sleep was needed during times of famine so as not to miss a hunting opportunity.
I might point out that there are many teenagers whose arousal thermostat is set too high, who have trouble sleeping, but are not overweight. While their leptin system is being challenged, it has not yet faltered. Their wired metabolism is burning all the calories they can eat and they may even have trouble gaining weight or keeping weight on. Such teens will be at high risk for becoming overweight later in life.
Many teens are not so lucky, and their leptin system has started faltering at an early age, not only causing obesity-related metabolic problems to lock into place but seriously compromising future health.
Current research clearly shows a lack of sleep in children is an independent risk factor for obesity, as poor sleepers have a 92% higher risk of obesity compared to the best sleepers. Researchers have also identified how a lack of sleep produces a stress response (cortisol excess) that also turns down fat burning and helps lock leptin problems into place.
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