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1. If you have herpes, your life isn’t over.
2. For those who don’t have herpes, HERPES IS NOT A PLAUGE
3. A safe, satisfying, and healthy sexual relationship can happen between someone who has herpes, and someone who doesn’t (condoms+ valtrex+ abstaining from sex when an outbreak is about to show up= 2 happy healthy people).
4. You should never settle for less because you have herpes.
5. Cold sores are HERPES, its called HSV 1
6. Herpes is contagious during an outbreak, and just before an outbreak when physical symptoms are just about to happen (never take chances with symptoms).
7. Herpes is as American as apple pie and baseball. Tons of people have it. You are not alone.
8. People who have herpes must be honest and tell their partners.
9. Herpes is not an STD, its a VIRUS.
10. People with herpes aren’t gross or broken. The worst part about having herpes is the social stigma attached to it. Maybe those who ignorantly reject those with herpes should do a little research and realize its very common. You probably already know people with genital herpes and cold sores who are too afraid to tell you because they fear your bad reaction!

For those with herpes…stop feeling ashamed, its a common thing. Speak openly about it, don’t let the ignorance of others make you feel bad. The more open you are about it, and the less you withdraw from society, the more you help to weaken the stigma.

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I found out my childhood cold sore virus had moved to my eye after a couple of misdiagnoses in 2003 – I was 29 at the time. Extreme light sensitivity, etc. in the high altitude dessert turned my life completely around. With all windows blacked out, I slept during the day and worked from home at night with an eye patch over my affected eye, which made detailed computer work pretty difficult. Other symptoms included, redness, pain, scratchiness, itchiness, blurred vision, runny eyes, general run down feeling, horrible anxiety and depression and as it progressed - I eventually got several flu like symptoms too (in bed for days at a time, fever, congestion, etc).

After several urgent care visits, I was finally diagnosed with eye herpes a month later by the hospital’s ophthamologist, he prescribed me viroptic and pred eye drops. I finally recovered after a couple months, but then I broke out again to the worst pain I’ve ever experienced. I cut my hair hoping that would stop the most severe headaches of my life. Doctor’s dismissed the feelings I had “in my head,” but eventually after a couple more doctors I got on oral anti-virals too (anti-virals have made an incredible difference!). It took many months to recover from that second outbreak, as I tried going off the drops and oral anti-virals, but I broke out again, and again. Finally we found some very knowledgeable and experienced doctors that got me off drops and on suppression therapy at UCLA’s Doris Stein’s Cornea Division. They have treated me like gold and I’ve gone there ever since.

Almost two years ago I moved a few hours outside of Chicago and I had only had a couple minor flare ups. We called my doctor in Cali and he said to take up to 2000mg of acyclovir if and when the virus acts up, and if it doesn’t clear right up to find a local ophthamologist. I had done very well up until very recently and I had a minor outbreak. Through a series of referrals I found another herpes specialist ophthamologist at the University of Illinois, Chicago. He confirmed my outbreak, switched me to Valtrex and I have another appointment with him in a couple weeks. I am grateful that when there is viral activity present, I now have control over it - I have become aware of what my body needs (usually an increase in anti-virals and a few days of rest/limited sun exposure), and I do well mostly on my own anymore.

I hadn’t done much of my own personal research since I first found out about my disease. I did a little online research, but mainly began therapy to get a better handle on my stress/anger issues. I was pretty frustrated there was limited information about ocular herpes, and with my first year of nothing but poor doctor advice, I didn’t want to further mislead myself with all the “miracle herpes cures” and whatnot out there. I recently did a great deal of research, though, and was pleased to find a little more info available, but I definitely had to dig past all the bogus hooey. Some of which I found was posts on herpes boards from frightened individuals – claims about brain herpes being a HUGE killer that we should all be afraid of it – that frightened me away from learning more about the very rare, extreme cases of my disease. I’ve since studied up on HSV-1 a great deal and I feel more confident that my life will go on even if I lose more vision and/or the virus keeps spreading. Actually when I read that many encephalitis (brain herpes) cases go undetected, because it is so difficult to detect, I wonder if I didn’t already have it when the headaches were so intense. All the other symptoms were there, and I was desperate enough to cut my long hair that I’d been growing out for over 10 years. I have lossed vision and at least two straight years of my life, living in a cave - herpes is not something to be taken lightly, but it’s also not something to be scared and run away from either. It is what it is, a pain in the… I think the best thing to do is find the best care for your body and don’t give up no matter what.

I find that generally people are scared to talk about herpes - but the majority of us have one type or another. People don’t know the dangers because they’re scared to get past the stigma or whatever; well I’ve been doing a lot of talking… Some of my family and friends seem scared of me right now for their own personal reasons, but that’s ok, my eye herpes brought about big changes, and that takes time to adapt to for people that don’t understand. (And, well, there are times when I don’t relate to them either… Because it’s difficult to go it alone and there aren’t always perfect support groups out there for rarer individual viral strains of herpes – I learned to seek out whatever support that’s available. I now have the best friends a gal could ever ask for and I’m always looking for more. I am so very grateful for those that have really been there for me. I look forward to being there for them too, ’cause I know I’m going to be just fine and it feels so wonderful to help those that have helped you. This is such a wonderfully informative site. Thank you Angela for your amazing contribution – I sure hope many others follow this shining example of getting the word out.

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Hand, foot and mouth disease is the most common cause of mouth sores in children, primarily in the age group from six months to three years old. It is caused by a group of viruses called enteroviruses, most commonly, the coxsackievirus A16.

Hand-foot-and-mouth disease isn’t related to foot-and-mouth disease (sometimes called hoof-and-mouth disease), which is a highly infectious viral disease found in farm animals. You can’t contract hand-foot-and-mouth disease from pets or other animals, and you can’t transmit it to them.

Hand, foot and mouth disease is a viral infection that can be caused by one of two viruses Coxsackie’s or Enteroviurs. This is characterized by blisters on the hands, feet, and mouth and in young children the buttocks is affected.

Hand, foot, and mouth disease is characterized by blisters of the hand, foot, and mouth. The blisters can be seen inside of the mouth on the cheeks, tongue, and gums.

The initial symptoms of hand, foot and mouth disease include fever, lack of appetite, sore throat and runny nose. A day or two after the initial symptoms appear, a blister-like rash forms on the hands, feet or mouth.

Coxsackie Virus - Read about Coxsackie virus treatment, infection during pregnancy, symptoms, causes and types (A, B). Type A causes herpangina, or hand, foot and mouth disease, while type B causes pleurodynia.

Twelve children ages 1 to 5 years and one adult with hand-foot-and-mouth disease were treated with oral acyclovir within one to two days of onset of the rash. Symptomatic relief, defervescence, and significant involution of lesions were seen with twenty-four hours of initiating therapy.

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Vaccination has changed little since the time of Louis Pasteur. The method involves deliberately injecting a dead or inactivate organism into a person to stimulate their immune system to produce cells that fight off the fully fledged organism.

However, many infections have resisted all attempts at producing vaccines. One of these is the herpes simplex virus type 2 (HSV-2), which is usually the cause of genital herpes.

Now Michal Margalith at Vical, a biopharmaceutical company based in San Diego, says he and colleagues have developed an HSV-2 vaccine using the emerging technology of DNA vaccines.

This involves injecting the patient with a circular piece of DNA called a plasmid that programs their cells to produce HSV-2 proteins that trigger an immune response. That should train the vaccinated person’s immune system to fight off the real virus.

The team says that the technique has successfully produced an immune response in mice. However, DNA vaccines are still highly experimental in humans.

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Drugs that combat herpes and CMV inhibit viral DNA replication by competing with viral substrates to form shorter, noneffective DNA chains. This action prevents replication of the virus, but it has little effect on the host cells of human because their DNA uses different substrate. These antiviral agents indicated for treatment of the DNA viruses herpes simplex, herpes zoster and CMV.

Contraindications and Cautions

Drugs indicated for the treatment of herpes and CMV should not used during pregnancy or lactation or I patients with known allergies to antiviral agents, renal disease which could interfere with excretion of the drug or severe CNS disorders.

Adverse Effects

The adverse effects most commonly associated with these antivirals include nausea and vomiting, headache and depression and rash and hair loss. Rash and inflammation and burning often occur at sites of IV injection and topical application.

Clinically Important Drug-Drug Interactions

The risk of nephrotoxicity increases when agents indicated for the treatment of herpes and CMV are used in combination with other nephrotoxic drugs. The risk of drowsiness also rises when these antiviral agents are taken with zidovudine and anti retroviral agent.

Nursing Diagnoses

The patient receiving a DNA-active antiviral agent may have the following nursing diagnoses related to drug therapy:
· Acute pain related to GI, CNS, local effects of drug
· Disturbed Sensory Perception related to CNS
· Deficient knowledge regarding drug therapy

Implementation

· Ensure good hydration to decrease the toxic effects on the kidneys.
· Administer the drug as soon as possible after the diagnosis has been made to improve effectiveness of the antiviral activity.
· Ensure that the patient takes the complete course of the drug regimen to improve effectiveness and decrease the risk of the emergence of resistant viruses.
· Wear protective gloves when applying the drug topically to decrease risk of exposure to the drug and inadvertent absorption.
· Provide safety precautions if CNS effects occur (e.g., use side rails, appropriate lighting, orientation, assistance) to protect the patient from injury.
· Warn the patient that GI upset, nausea, and vomiting can occur, to prevent undue anxiety and increase awareness of the importance of nutrition.
· Monitor renal function tests periodically during treatment to detect and respond to renal toxicity as soon as possible.
· Provide the patient with instructions about the drug therapy and to promote compliance.

The patient should:

· Avoid sexual intercourse if genital herpes is being treated, because these drugs do not cure the disease.
· Wear protective gloves when applying topical agents.
· Avoid the driving and hazardous tasks dizziness and drowsiness occurs.

Evaluation

· Monitor patient response to the drug
· Monitor for adverse effects
· Evaluate effectiveness of the teaching plan
· Monitor for the effectiveness of comfort and safety measures and compliance with regimen

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Herpes viruses account for a broad range of conditions, including cold sores, encephalitis, shingles and genital infections. Cytomegalovirus (CMV) although slightly different from the herpes virus can affect the eye, respiratory tract and liver and reacts to many of the same drugs.

1. Acyclovir (Zovirax) – is specific for herpes virus infections. Is excreted unchanged in the urine and therefore must be used cautiously in the presence of renal impairment. It crosses into breast milk and exposes the neonate to high levels of the drug.
2. Cidofovir (Vistide) – is used to treat CMV retinitis in patients with AIDS only. It is associated with severe renal toxicity and granulocytopenia. It is excreted unchanged in the urine and must be given with probenecid to increase renal clearance of the drug. Use in children with AIDS should be very cautious because of potential.
3. Famiciclovir (Famvir) – is most effective in treating herpes infections. It is well absorbed in the GI tract, reaching peak levels in 2 to 3 hours. It is metabolized in the liver and excreted in the urine and feces.
4. Foscarnet (Foscavir) – is available in intravenous (IV) form only. It can be highly toxic to the kidneys and is reserved for treatment of CMV retinitis in immunocompromised patients and for mucocutanneous acyclovir-resistant herpes simplex infections.
5. Ganciclovir (Cytovene) – which is available in IV and oral forms and is used for long term treatment and prevention of CMV infections. Primarily excreted unchanged in the feces with some urinary excretion. It is carcinogenic and should be used only with extreme caution in children.
6. Valacyclovir (Valtrex) – is an oral agent used for the treatment of herpes zoster and recurrent genital herpes. Rapidly absorbed from the GI tract and metabolized in the liver to acyclovir. Excretion occurs through the urine so caution should be used in patients with renal impairment.
7. Valganiclovir (Valcyte) – which is the oral prodrug of ganciclovir, is used for the treatment of CMV retinitis in AIDS patients. Primarily excreted unchanged in the feces with some urinary excretion.

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Herpes simplex is part of a group of other herpes viruses called Herpesviridae, that include human herpes virus 8 (the cause of Kaposi’s sarcoma), herpes zoster (the virus responsible for shingles and chicken pox) and cytomegalovirus (CMV).

There are two strains of the virus: type 1 (HSV1), usually associated with the mouth, lip, and face infections and is responsible for cold sores and fever blisters; and type 2 (HSV 2), usually associated with infections of the sex organs (it is the main cause of the condition known as genital herpes) and both of these strains of virus can cause extremely painful infections in humans.

Both type 1 and type 2 herpes viruses reside in a latent state in the nerves which supply sensation to the skin. The virus ‘buds off’ from the nerve ending and infects the surrounding skin cells, producing a painful cluster of pale blisters that are crammed with herpes simplex virus and are highly infectious.

Recurrences of Type 1 infection can occur on virtually any part of the body surface but are most frequent on the face, particularly on or around the lips. Infection of the mouth is very common and though the appearance may be a source of concern, it presents no serious risks to your general health.

Herpes simplex is actually the most common virus found in humans, causing infections of the skin and mucous membranes but is also an uncommon cause of more serious infections in other parts of the body. Virus infections from herpes may cause severe extensive disease in immune-suppressed individuals (HIV or cancer patients, for example).

Like all members of the herpes virus family, the herpes simplex virus stays with us for life. The virus affects only humans and there is no cure. It is estimated that over half a million new cases of the virus occur every year and in the United States alone, one out of five of the total adolescent and adult population are infected.

Newborns can catch herpes simplex from their mother during birth. If a newborn is infected with the virus, the following symptoms of infection may emerge during the first month after birth: irritability, breathing problems, fever, convulsions, jaundice (yellowish skin), sores on the skin, bruising or low platelet counts (platelets are the part of blood that makes it clot). In the most extreme cases, babies infected with herpes simplex may develop severe eye problems, a small brain size, mental retardation, seizures or even die.

The virus rarely spreads to the baby through the placenta of the mother but to decrease the risk of infecting newborns, a caesarean delivery (C-section) is recommended for pregnant women who have an active herpes simplex infection at the time of delivery.

One fascinating fact about herpes is that at least one third of children are infected by the virus by the end of childhood.

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There are herpes drugs, which help with the frequency of shedding (the warts). Unfortunately you can shed at any time, but the medication does help. Statistically speaking, after five years with a partner who has herpes you’re most likely going to be infected. That doesn’t mean you’re going to have lesions, but if you do blood tests of a person who has lesions, most people have it.

There are two types of herpes. The ones that we look out for are herpes simplex, type I and type II. Type I is from the waist up and type II is the genital herpes from the waist down. You can inoculate yourself by just touching your mouth and then touching the genitalia. You can get type I in the genitalia area and then get type II in the oral area. If someone has it on the mouth you can touch someone down there and contract it. Many times, people have one outbreak and then only a little bump, but then they’re shedding and they don’t realize it.

What can cause outbreaks and how do you contain them?

Full outbreaks are caused by stress, trauma or emotional upsets. Some people will outbreak every month and then other people will have just one outbreak. The longer you have herpes, the less frequent outbreaks are. Keeping the infected area dry is important; it will run its course. There are ointments and stuff some people swear by; other people say they don’t do a thing.

Besides pregnancy, what kind of emotional toll can sex take on a young person?

We see the implications of sex before marriage every day. What we tell the girls that come through our doors is that premarital sex is always negative; every single consequence is negative. It’s because they are dying to have somebody love them. A lot of times it’s because their father never married, or they are fatherless.

There is a huge emotional component to young women having sex. They desperately want a man to take care of them. Women give sex to get love, and men give love to get sex. Sex is supposed to be a fabulous bonding thing for marriage, and it is degraded outside of marriage, and then it becomes not the special thing God intended it to be.

Also, STDs are transferable every day of the year - herpes, HIV, HPV and hepatitis, which you will have for life. One woman had contracted herpes and she could barely walk on her wedding day. That’s just a degrading way to enter a marriage. These relationships tend not to be real stable in spite of the sex; they haven’t built it on a strong friendship so it’s shaky and when they split up there is a lot of emotional pain. So it’s just humiliating and sad.

What about the emotional impact on a guy?

Guys are just as vulnerable to family instability and they need a father as well, and a lot of this desire is to keep this girl, and then jealousy comes in. They want stability as well. Men do have a much more powerful sex drive than women. Women don’t feel pressure to have sex but men are always building up sperm. But they have the same emotional needs; they have that component as well. We get some guys who are very broken hearted as well. They’re not animals either, but some are and can be pretty callous as well. But most guys care about the girl, and if she’s pregnant, it’s a pregnant experience for both the male and the female. Strong parenting and loving married parents are the best a child could have. Encourage your readers to get married and make it last.

In your opinion, how big of a difference does it make to wait until you are married? Does having a certificate make that big of a difference?

Huge. We are in a culture that down-plays it and says, “It’s ridiculous to be concerned about it.” The impact is far more devastating and very glossed over. For people to put God first, speak his will and make a commitment is huge. Wait until you are older and ready to get married and are able to really start focusing on whom God is bringing into your life. It’s almost irresistible apart from God’s grace. We just crave that intimacy, but that’s why God created the institution of marriage. I think premarital sex interferes with peoples’ ability to do well and focus in college.

Do you think that there are still couples or individuals out there who are waiting for marriage, or is the number of sexually active youth increasing all the time?

We see a population that is definitely not waiting. On the Whitworth campus I’m sure there are people who lie about it, but there are definitely people who are waiting.

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Q: I read that in the United States over 6 million men and women get a Human Papilloma virus (HPV) infection every year. If it’s so common, should we really be worried?

Well, they found that cervical cancer is related to a virus, and they’ve finally been able to document cervical cancer: It’s mostly due to HPV. There are hundreds of types of the HPV virus. There are several different types of genital warts, and half of them are benign. Usually, most warts have their own lifespan and will eventually go away. The other half are the higher risks ones and are known to cause cervical cancer.

Gardasil does not protect against all of these viruses. The ones that they do protect you against are the high-risk viruses. For the benign types, they protect you against two of the viruses. What’s really interesting is that they are also finding out that you get some sort of cross protection. The way the virus lives is sort of like a branch on the tree - if you immunize against this one you might have cross protection against the second one on the branch. You’ll be protected against eight types of the virus instead of four.

Q: I’ve heard that HPV is untraceable in men. Is this true?

Pretty much, unless they have warts. I don’t think there are any blood tests to detect HPV in men. But they are trying to get our young men vaccinated, too. They are working on a Gardasil for men. They are figuring if they can immunize young men and young girls before they get sexually active they can cut down cervical cancer just by doing that.

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